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Although aldosterone-induced oxidative or nitrative stress quickly stimulated antioxidant or detoxifying enzymes such SOD, thioredoxin (TRX), HO-1, or GCSc, this adaptive survival response appeared to be fleeting and overpowered by a chronic increased generation of ROS/RNS

Supporting these results, the administration of mitoperoxidase 2 in the mice model 24 h after WBI (-rays, 9 Gy) offered a significant increase in the 30-day survival of mice

It is intended strictly for in-vitro and preclinical laboratory research, not for human or veterinary use