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The pathological effects of oxidative stress underscore the importance of developing therapeutic strategies aimed at reducing ROS levels and enhancing antioxidant defenses to improve male fertility outcomes

[60] studied the effects of GHK-Cu both in vitro (lipopolysaccharide-induced RAW 264.7 macro-phages) and in vivo (acute lung injury in mice)

These variants may influence individual glutathione capacity and oxidative stress susceptibility, though clinical expression is modified by diet, exercise, and environmental factors