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Finally, newly developed GEMM models, which are only partially leveraged here for their stable Keap1 suppressed status, will require further examination to better understand the effects of chronic cigarette smoke exposure and development of chemoradiotherapy sensitivity and/or resistance

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In summary, several aspects of the proposed mechanism must be confirmed, including whether burden of citrullination of the lung drives ACPA generation and if Th17 cells are enhanced in the joint to drive neutrophilia